Search results for " Atrophic"

showing 10 items of 19 documents

TLR1 and PRKAA1 Gene Polymorphisms in the Development of Atrophic Gastritis and Gastric Cancer.

2018

Background & Aims: Previous genome-wide association studies showed that genetic polymorphisms in toll-like receptor 1 (TLR1) and protein kinase AMP-activated alpha 1 catalytic subunit (PRKAA1) genes were associated with gastric cancer (GC) or increased Helicobacter pylori (H. pylori) infection susceptibility. The aim of this study was to evaluate the association between TLR1 and PRKAA1 genes polymorphisms and H.pylori infection, atrophic gastritis (AG) or GC in the European population.Methods: Single-nucleotide polymorphisms (SNPs) were analysed in 511 controls, 340 AG patients and 327 GC patients. TLR1 C>T (rs4833095) and PRKAA1 C>T (rs13361707) were genotyped by the real-time po…

0301 basic medicineAdultGastritis AtrophicMalemedicine.medical_specialtyAtrophic gastritisSingle-nucleotide polymorphismAMP-Activated Protein KinasesGastroenterologyPolymorphism Single NucleotideWhite Peoplelaw.inventionHelicobacter Infections03 medical and health sciences0302 clinical medicineGene FrequencylawRisk FactorsStomach NeoplasmsInternal medicineGenotypemedicineSNPHumansGenetic Predisposition to DiseaseAllelePolymerase chain reactionGenetic Association StudiesGenetic associationAgedbiologyHelicobacter pyloribusiness.industryGastroenterologyHelicobacter pyloriMiddle Agedbiology.organism_classificationmedicine.diseaseToll-Like Receptor 1Europe030104 developmental biologyPhenotype030220 oncology & carcinogenesisCase-Control StudiesFemalebusinessJournal of gastrointestinal and liver diseases : JGLD
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Association of Long Non-Coding RNA Polymorphisms with Gastric Cancer and Atrophic Gastritis

2020

Long non-coding RNAs (lncRNA) play an important role in the carcinogenesis of various tumours, including gastric cancer. This study aimed to assess the associations of lncRNA ANRIL, H19, MALAT1, MEG3, HOTAIR single-nucleotide polymorphisms (SNPs) with gastric cancer and atrophic gastritis. SNPs were analyzed in 613 gastric cancer patients, 118 patients with atrophic gastritis and 476 controls from three tertiary centers in Germany, Lithuania and Latvia. Genomic DNA was extracted from peripheral blood leukocytes. SNPs were genotyped by the real-time polymerase chain reaction. Results showed that carriers of MALAT1 rs3200401 CT genotype had the significantly higher odds of atrophic gastritis …

0301 basic medicineGastritis AtrophicMalemedicine.medical_specialtylcsh:QH426-470GenotypeAtrophic gastritisSingle-nucleotide polymorphismmedicine.disease_causeGastroenterologyPolymorphism Single NucleotideArticleTertiary Care Centers03 medical and health sciences0302 clinical medicineGene FrequencyStomach NeoplasmsInternal medicineGermanyatrophic gastritisGenotypeGeneticsmedicineOdds RatioHumansGenetic Predisposition to DiseaseRNA NeoplasmGenetics (clinical)AllelesAgedMALAT1long non-coding RNAbusiness.industrylong non-coding RNA ; single-nucleotide polymorphism ; gastric cancer ; atrophic gastritisgastric cancerCancerHOTAIRMiddle Agedsingle-nucleotide polymorphismmedicine.diseaseLong non-coding RNAlcsh:Genetics030104 developmental biology030220 oncology & carcinogenesisFemaleRNA Long NoncodingCarcinogenesisbusinessGenes
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Allopurinol partially prevents disuse muscle atrophy in mice and humans

2018

AbstractDisuse muscle wasting will likely affect everyone in his or her lifetime in response to pathologies such as joint immobilization, inactivity or bed rest. There are no good therapies to treat it. We previously found that allopurinol, a drug widely used to treat gout, protects muscle damage after exhaustive exercise and results in functional gains in old individuals. Thus, we decided to test its effect in the prevention of soleus muscle atrophy after two weeks of hindlimb unloading in mice, and lower leg immobilization following ankle sprain in humans (EudraCT: 2011-003541-17). Our results show that allopurinol partially protects against muscle atrophy in both mice and humans. The pro…

0301 basic medicineProteasome Endopeptidase Complexmedicine.medical_specialtyScience[SDV]Life Sciences [q-bio]Allopurinolmedicine.medical_treatmentAllopurinolHindlimbBed restArticleMice03 medical and health sciences0302 clinical medicineAtrophyPhysical Conditioning AnimalInternal medicineAnimalsHumansMedicineAnkle InjuriesMuscle SkeletalWastingSoleus muscleMultidisciplinaryUbiquitinbusiness.industryQRmedicine.diseaseMuscular Disorders AtrophicMuscle atrophy3. Good healthGoutMuscular Atrophy030104 developmental biologyEndocrinologyHindlimb SuspensionMedicinemedicine.symptombusiness030217 neurology & neurosurgerymedicine.drugScientific Reports
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Prevalence of Atrophic Gastritis in Kazakhstan and the Accuracy of Pepsinogen Tests to Detect Gastric Mucosal Atrophy

2019

Background Atrophic gastritis is considered precursor condition for gastric cancer. There is so far limited evidence on the performance of pepsinogens for atrophy detection in Central Asia. The aim of our study was to detect the prevalence of atrophic gastritis in the asymptomatic adult population in Kazakhstan as well as address the accuracy of pepsinogen testing in atrophy detection. Methods Healthy individuals aged 40-64 were included. Upper endoscopy and pepsinogens (PG) evaluation were performed. PG were analysed in plasma by latex agglutination. Cut off values were used to define decreased PG values (PGR ≤ 3 and PG I ≤ 70 ng/mL); severely decreased PG values (PGR ≤ 2 and PG I ≤ 30 ng/…

AdultGastritis AtrophicMale0301 basic medicinemedicine.medical_specialtyAtrophic gastritisPopulationgastricAsymptomaticGastroenterology03 medical and health sciences0302 clinical medicineAtrophyatrophyPepsinPepsinogen AInternal medicinePrevalenceHumansMedicineeducationeducation.field_of_studyPepsinogensbiologyatrophicbusiness.industrygastric cancerscreeninggastritisEndoscopyGeneral MedicineGold standard (test)Middle AgedPrognosismedicine.diseaseKazakhstanLatex fixation test030104 developmental biologyROC CurveGastric Mucosa030220 oncology & carcinogenesisbiology.proteinFemaleGastritismedicine.symptombusinessResearch ArticleFollow-Up StudiesAsian Pacific Journal of Cancer Prevention
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Lack of association between gene polymorphisms of Angiotensin converting enzyme, Nod-like receptor 1, Toll-like receptor 4, FAS/FASL and the presence…

2011

Abstract Background Several polymorphisms of genes involved in the immunological recognition of Helicobacter pylori and regulating apoptosis and proliferation have been linked to gastric carcinogenesis, however reported data are partially conflicting. The aim of our study was to evaluate potential associations between the presence of gastric cancer (GC) and high risk atrophic gastritis (HRAG) and polymorphisms of genes encoding Angiotensin converting enzyme (ACE), Nod-like receptor 1 (NOD1), Toll-like receptor 4 (TLR4) and FAS/FASL. Methods Gene polymorphisms were analyzed in 574 subjects (GC: n = 114; HRAG: n = 222, controls: n = 238) of Caucasian origin. ACE I/D (rs4646994), NOD1 796G>…

AdultGastritis AtrophicMaleFas Ligand ProteinGenotypeAtrophic gastritisPeptidyl-Dipeptidase AWhite PeopleFas ligandHelicobacter InfectionsRisk FactorsStomach NeoplasmsNod1 Signaling Adaptor ProteinNOD1GenotypemedicineGeneticsHumansGenetics(clinical)fas ReceptorAllelesGenetics (clinical)AgedAged 80 and overPolymorphism GeneticHelicobacter pyloribiologyCancerAngiotensin-converting enzymeMiddle AgedHelicobacter pylorimedicine.diseasebiology.organism_classificationToll-Like Receptor 4ApoptosisImmunologybiology.proteinFemalePrecancerous ConditionsResearch ArticleBMC Medical Genetics
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Different Pattern of Inflammatory and Atrophic Changes in the Gastric Mucosa of the Greater and Lesser Curvature.

2015


 
 
 
 
 
 Background & Aims: Appropriate biopsy sampling is important for the classification of gastritis, yet the extent of inflammation and atrophy of different regions of the stomach with chronic gastritis have been addressed only in a few studies. The aim of our study was to analyze the inflammatory, atrophic and metaplastic changes in the greater and lesser curvature of the antrum and corpus mucosa.
 Methods: 420 patients undergoing upper endoscopy were enrolled in the study. Four expert gastrointestinal pathologists graded biopsy specimens according to the updated Sydney classification.
 Results: The obtained results showed that the mononu…

AdultGastritis AtrophicMalePathologymedicine.medical_specialtyBiopsyChronic gastritisSeverity of Illness IndexAtrophyPredictive Value of TestsBiopsymedicinePyloric AntrumHumansProspective StudiesAntrumAgedObserver VariationMetaplasiamedicine.diagnostic_testHelicobacter pyloribusiness.industryStomachGastroenterologyIntestinal metaplasiaReproducibility of ResultsAnatomyMiddle Agedmedicine.diseaseCurvatures of the stomachmedicine.anatomical_structureGastric MucosaFemaleGastritismedicine.symptombusinessGranulocytesJournal of gastrointestinal and liver diseases : JGLD
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Prevalence of Helicobacter pylori infection and atrophic gastritis in Latvia.

2012

Helicobacter pylori infection and atrophic gastritis are related to an increased risk for gastric cancer. There is a decrease in global H. pylori prevalence. We analyzed the prevalence of H. pylori infection in Latvia by the plasma IgG test and the presence of atrophy by means of pepsinogen testing.This subanalysis was carried out on a randomly selected cross-sectional sample of a general population of adults to access cardiovascular risk factors. Plasma samples were screened for H. pylori IgG (cutoff value 24 U/ml), and pepsinogens (Pg) I and II. Pg cutoff values of PgI/PgII ≤ 3 and PgI ≤ 70 ng/ml were used to assess the prevalence of atrophy of any grade and PgI/PgII ≤ 2 and PgI ≤ 30 ng/m…

AdultGastritis AtrophicMalemedicine.medical_specialtyHelicobacter pylori infectionAdolescentCross-sectional studyAtrophic gastritisGastroenterologySeverity of Illness IndexHelicobacter InfectionsYoung AdultAtrophySex FactorsPredictive Value of TestsRisk FactorsStomach NeoplasmsInternal medicinePepsinogen AmedicinePepsinogen CPrevalenceHumansYoung adultAgedAged 80 and overChi-Square DistributionHepatologybiologyHelicobacter pyloribusiness.industryGastroenterologyAge FactorsCancerMiddle Agedbacterial infections and mycosesmedicine.diseaseAntibodies BacterialLatviaCross-Sectional Studiesbiology.proteinLinear ModelsFemaleGastritismedicine.symptomAntibodybusinessPrecancerous ConditionsBiomarkersEuropean journal of gastroenterologyhepatology
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The phenotype of gastric mucosa coexisting with Barrett's oesophagus.

2001

Barrett's oesophagus complicates the gastro-oesophageal acid reflux. Helicobacter pylori infection, particularly with cagA positive strains, induces inflammatory/atrophic lesions of the gastric mucosa, which may impair acid output. No systematic study has investigated the phenotype of the gastric mucosa coexisting with Barrett's oesophagus. This study was designed to identify the phenotype of gastric mucosa associated with Barrett's oesophagus.In this retrospective case control study, the phenotype of the gastric mucosa was histologically characterised in 53 consecutive patients with Barrett's oesophagus and in 53 (sex and age matched) non-ulcer dyspeptic controls. Both patients and control…

AdultGastritis AtrophicMalemedicine.medical_specialtyPathologyAtrophic gastritisBiopsySpirillaceaeBarrett's oesophagus gastritis in Barrett's oesophagus Barrett's oesophagus and gastric precancerous lesionsdigestive systemGastroenterologyHelicobacter InfectionsPathology and Forensic MedicineBarrett's oesophagus and gastric precancerous lesionsBarrett EsophagusInternal medicineotorhinolaryngologic diseasesGastric mucosamedicineHumansCagAAgedRetrospective StudiesAged 80 and overHelicobacter pyloribiologybusiness.industrygastritis in Barrett's oesophagusStomachdigestive oral and skin physiologyIntestinal metaplasiaGeneral MedicineMiddle AgedHelicobacter pyloribiology.organism_classificationmedicine.diseasedigestive system diseasesBarrett's oesophagussurgical procedures operativePhenotypemedicine.anatomical_structureGastric MucosaCase-Control StudiesPapersFemaleGastritismedicine.symptombusiness
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Gastric plasma biomarkers and Operative Link for Gastritis Assessment gastritis stage

2011

Introduction The Operative Link for Gastritis Assessment (OLGA) staging system has been proposed as a histopathological reporting system of gastric atrophy. Noninvasive methods for indirect evaluation of gastric mucosal atrophy by biomarkers are also being introduced. Objectives To analyze gastric mucosal atrophy by biomarkers, pepsinogen I (PgI), pepsinogen II (PgII), PgI/PgII ratio, fasting gastrin-17 (G-17), stimulated gastrin-17 (sG-17), in relation to OLGA gastritis stage. Patients and methods Gastric biopsies were taken from 269 prospective patients referred for upper endoscopy because of dyspeptic problems and evaluated by two expert pathologists (D.J. and P.S.). Atrophy was assessed…

AdultGastritis AtrophicMalemedicine.medical_specialtyStage iiPlasma biomarkersGastroenterologyHelicobacter InfectionsAtrophyPepsinogen AInternal medicineGastrinsPepsinogen CHumansMedicineProspective StudiesStage (cooking)Prospective cohort studyAgedAged 80 and overHelicobacter pyloriHepatologybusiness.industryGeneral surgeryGastroenterologyMiddle Agedmedicine.diseaseFemaleGastritismedicine.symptombusinessGastric mucosal atrophyReporting systemBiomarkersEuropean Journal of Gastroenterology & Hepatology
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Analysis of thiamine transporter genes in sporadic beriberi

2014

Abstract Objective Thiamine or vitamin B 1 deficiency diminishes thiamine-dependent enzymatic activity, alters mitochondrial function, impairs oxidative metabolism, and causes selective neuronal death. We analyzed for the first time, the role of all known mutations within three specific thiamine carrier genes, SLC19 A2, SLC19 A3 , and SLC25 A19 , in a patient with atrophic beriberi, a multiorgan nutritional disease caused by thiamine deficiency. Methods A 44-year-old male alcoholic patient from Morocco developed massive bilateral leg edema, a subacute sensorimotor neuropathy, and incontinence. Despite normal vitamin B 1 serum levels, his clinical picture was rapidly reverted by high-dose in…

AdultMalemedicine.medical_specialtySLC19 A- SLC25 A19SLC19 AEndocrinology Diabetes and MetabolismGene mutationBeriberimedicine.disease_causeMitochondrial Membrane Transport Proteinslaw.inventionBeriberilawInternal medicineGenotypemedicineThiamine transporterObjective: Thiamine or vitamin B1 deficiency diminishes thiamine-dependent enzymatic activity alters mitochondrial function impairs oxidative metabolism and causes selective neuronal death. We analyzed for the first time the role of all known mutations within three specific thiamine carrier genes SLC19 A2 SLC19 A3 and SLC25 A19 in a patient with atrophic beriberi a multiorgan nutritional disease caused by thiamine deficiency. Methods: A 44-year-old male alcoholic patient from Morocco developed massive bilateral leg edema a subacute sensorimotor neuropathy and incontinence. Despite normal vitamin B1 serum levels his clinical picture was rapidly reverted by high-dose intramuscular thiamine treatment suggesting a possible genetic resistance. We used polymerase chain reaction followed by amplicon sequencing to study all the known thiamine-related gene mutations identified within the Human Gene Mutation Database. Results: Thirty-seven mutations were tested: 29 in SLC19 A2 6 in SLC19 A3 and 2 in SLC25 A19. Mutational analyses showed a wild-type genotype for all sequences investigated. Conclusion: This is the first genetic study in beriberi disease. We did not detect any known mutation in any of the three genes in a sporadic dry beriberi patient. We cannot exclude a role for other known or unknown mutations in the same genes or in other thiamine-associated genes in the occurrence of this nutritional neuropathy.HumansThiamineGenePolymerase chain reactionGeneticsMutationNutrition and DieteticsbiologyMembrane Transport ProteinsThiamine Deficiencymedicine.diseaseAlcoholismEndocrinologyMutationbiology.proteinThiamineMutations
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